Restenosis After Balloon Valvuloplasty in Dogs with Congenital Pulmonary Stenosis
نویسندگان
چکیده
The aims of this study were to reveal the prevalence of complications associated with PBV over a long-term follow-up, and to investigate the factors related to those complications, especially pulmonary restenosis. Forty dogs who underwent PBV were retrospectively reviewed. Long-term follow-up was available in 22 dogs. Exacerbation of PI and TR after PBV was observed in some dogs; however, right-sided heart failure due to PI or TR was not observed in any of them. During long-term followup, restenosis developed in 6 dogs (6/22). Restenosis was noticed 5.5 months (ranging from 1.5 to 68.2 months) after successful PBV. Multiple regression analysis identified atenolol administration prior to PBV as the independent predictor of restenosis during long-term follow-up. Further investigation is warranted to investigate the mechanism of restenosis and to determine a strategy to prevent complications. INTRODUCTION Pulmonary stenosis (PS) is one of the most common congenital heart defects (CHD) in dogs.1 Pulmonary valvular stenosis (PVS) is the most common form of PS.1, 2 When PS is severe, the patient may develop syncope, right-sided congestive heart failures, or sudden death.3-6 Pulmonary balloon valvuloplasty (PBV) has been known to be effective for valvular stenosis, and is considered the first-line treatment for severe PVS in humans and dogs.4, 5, 7, 8 According to previous literature of human and dogs, complications of PBV after successful reduction of PG included restenosis of pulmonary valve, tricuspid valve regurgitation (TR), and pulmonary valve insufficiency (PI). 3-5, 9-12 Restenosis was noticed in some patients during longterm follow-up after PBV despite decreasing of the peak instantaneous pressure gradient across the pulmonary valve (PG) at immediate follow-up after PBV.3-5,9,10 To the best of author’s knowledge, there has been no report in veterinary medicine focusing on the Restenosis After Balloon Valvuloplasty in Dogs with Congenital Pulmonary Stenosis
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